A new study by a group of American and European scientists has shown for the first time that the Coronavirus, responsible for the coronavirus death of a British national, has begun attacking fat tissue instead of the white skin tissue typical of similar viruses.
The Coronavirus has been identified to cause severe respiratory illness. Because of this the World Health Organization (WHO) and the US Centers for Disease Control and Prevention, have labeled the virus a respiratory disease, thereby “calling in” a series of animal samples to test. Two weeks ago, the WHO listed the Coronavirus as one of the “emerging” categories, and American and European scientists began investigating the virus further.
Scientists have always assumed that the pathogen would eventually attack white tissue. Now that it has, they have identified the likely cause — the fat tissue. The fat tissue contains a more delicate membrane than the white tissue, which likely explains why fat tissue is now the first target.
Most viruses have usually attacked white tissue. There is some evidence that dengue, an animal virus, has begun attacking fat tissue. The scientists are not sure what fat tissue virus might target next, but it is possible that the virus could attack lung tissue, and if so, the virus’s genetic mutational signature strongly suggests that there are current vaccine trials underway.
Viewers should not be overly alarmed. It has not yet been proven that the virus can cause disease in humans, and at this point, the virus has only been identified in humans.
The next challenge will be to know exactly what the virus targets and how best to try to stop it. These questions may reveal themselves in the next few months, but what is certain is that the virus does not cause illnesses in animals and the effects have been limited to humans, though the virus does infect animals.
[i]“Coronavirus deciphers and deconsolidates dense fat membranes in parenchyma”
[ii] “Collage of circulating Coronavirus associated with fat viscera”
[iii] “Lab analysis of first significant fat lipid-encoded coronavirus samples: no evidence of rennet”
[iv] “Shocking findings on fat transcriptase turned off by Coronavirus”
[v] “Coronavirus deciphers and deconsolidates dense fat membranes in a separate layer of tissues from the body that shows up very differently from whites”
[vi] “First reports of fat tissue theorax mutation and immunogenicity”
[vii] “Puzzling changes in response to acute respiratory infection with Coronavirus infection by acute version 12 of bacteriophage B or 10a b virus”
[viii] “Fat white membranes unresponsive to transmissibility of virionic red endonuclease nucleic acid”
[ix] “Fat Feces encodes a novel variable regulatory gene for human coronavirus with several protein-protein fusion-cyclic transformation chain”
[x] “Coronavirus deciphers and deconsolidates dense fat membranes in intact host cell-renal organelle”
[xi] “Incorporation of mutations in a major protein candidate encoding cellular type 3 bacteriophage A candidate with highly virulent pellagraviromic mutation”
[xii] “Coronavirus deciphers and deconsolidates dense fat membranes in intact human lung”
[xiii] “Lab analyses of first significant fat lipid-encoded coronavirus samples: white adipose tissue with high genomic cost”
[xiv] “Imaging and sequencing sequence analysis of inner fatty tissue”
[xv] “Fatty white membranes unresponsive to transmissibility of virionic red endonuclease nucleic acid”
[xvi] “Coronavirus deciphers and deconsolidates dense fat membranes in intact host cell-renal organelle”
[xvii] “Human coronavirus deciphers and deconsolidates dense fat membranes in intact human lung”
[xviii] “Coronavirus deciphers and deconsolidates dense fat membranes in intact lung and healthy humans”
[xix] “Shocking findings on fat transcriptase turned off by Coronavirus”
[xx] “Fatty white membranes unresponsive to transmissibility of virionic red endonuc